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Elevated levels of reactive oxygen species (ROS) in osteoarthritic joints can directly damage proteins, lipids, and nucleic acids, triggering several cell death pathways, including apoptosis, pyroptosis, and autophagy-related cell death (16)

doi: 10.1016/j.numecd.2012.11.006 173 FintiniDGrugniGBocchiniSBrufaniCDi CandiaSCorriasAet al

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Ex is known to decrease the homoeostatic drive, i.e., hunger, to a level so low that is known to weaken or eradicate hedonic contrast effects [33, 37, 38]

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